@article{oai:miyazaki-u.repo.nii.ac.jp:00000858,
 author = {池田, 正浩 and Ikeda, Masahiro and Prachasilchai, Worapat and Burne-Taney, Melissa J. and Rabb, Hamid and Yokota-Ikeda, Naoko},
 issue = {7-8},
 journal = {Drug discovery today, DDT},
 month = {Apr},
 note = {Acute renal failure (ARF) is a common cause of mortality and morbidity in hospitalized patients. Ischemia is an important cause of ARF and ARF due to ischemic injury is traditionally referred to as ischemic acute tubular necrosis (ATN). There is growing evidence that ischemic ATN is associated with intra-renal inflammation using ischemic ATN models. Consequently, intra-renal inflammation is an attractive target for the development of novel drug therapies for ARF. In this review, we will outline ischemic ATN models, the pathophysiogical roles of inflammatory cells such as T and B cells in ischemic ATN models, and effective T and B cell therapeutic reagents.},
 pages = {364--370},
 title = {Ischemic acute tubular necrosis models and drug discovery: a focus on cellular inflammation},
 volume = {11},
 year = {2006},
 yomi = {イケダ, マサヒロ}
}