@article{oai:miyazaki-u.repo.nii.ac.jp:00003337,
 author = {村上, 隆之 and Murakami, Takayuki and 斎藤, 勇夫 and Saito, Isao and 野坂, 大 and Nosaka, Dai and 立山, 晉 and Tateyama, Susumu and 中野, 英治 and 芦沢, 広三 and Ashizawa, Hirozo and 浜名, 克己 and Hamana, Katsumi and 幡谷, 正明 and Hataya, Masaaki and Nakano, Eiji and 幡谷, 正明 and Hataya, Masaaki},
 issue = {1},
 journal = {宮崎大学農学部研究報告, Bulletin of the Faculty of Agriculture, Miyazaki University},
 month = {Oct},
 note = {生後9ヵ月で未経産牛乳房炎に罹患した25ヵ月齢の末経産牛の乳房1例と, 妊娠5ヵ月で本症に罹患した分娩後の乳房2例を用いて,本症における分娩後無乳症の病理発生について検索した.
 罹患分房は,治療後,健康分房と等しく発育し,分娩時には乳分泌像を示す腺胞が充分発達している. しかし,いずれの罹患分房も乳管洞が存在せず,結合組織に置換されていた.
 したがって,末経産牛乳房炎における分娩後の無乳症は,乳管洞の炎症性破壊による消失の結果発生すると考えられる., The pathogenesis of postpartum agalactia was investigated on three cases of heifer mastitis. Case No.1 was a udder obtained from non-pregnant heifer of 25 months of age which was suffered from mastitis at 9 months of age. Case No. 2 and No. 3 were udders obtained from postpartum cows both of which were suffered from mastitis at 5 months of pregnancy.
    All the affected quarters were clinically recovered as a result of quick and complete treatment. Thereafter, they had well developed as same as other normal quarters. Post-partum cases showed the well developed alveoli which meaned the active milk secretion at parturition. However, all the affected quarters became agalatia, because they had no true lactiferous sinus. The portion fit for lactiferous sinus was filled with connective tissue.
    It is suggested that the inflammatory destruction and disappearance of lactiferous sinus occured by heifer mastitis is a main reason to cause the postpartum agalactia.},
 pages = {45--51},
 title = {未経産牛乳房炎に関する研究 (第15報) : 分娩後無乳症の病理発生},
 volume = {26},
 year = {1979},
 yomi = {ムラカミ, タカユキ and サイトウ, イサオ and ノサカ, ダイ and タテヤマ, ススム and ナカノ, エイジ and アシザワ, ヒロゾウ and ハマナ, カツミ and ハタヤ, マサアキ and ハタヤ, マサアキ}
}